People almost always have palpitations and other symptoms of heart failure (for example, breathlessness, chest discomfort, and/or fainting).
Electrocardiography is used to make the diagnosis.
Drugs and procedures to destroy abnormal areas of the ventricles may be used, but usually an automatic implantable defibrillator is required.
(See also Overview of Abnormal Heart Rhythms.)
Ventricular tachycardia may be thought of as a sequence of consecutive ventricular premature beats. Sometimes only a few such beats occur together, and then the heart returns to a normal rhythm. Ventricular tachycardia that lasts more than 30 seconds is called sustained ventricular tachycardia.
Sustained ventricular tachycardia usually occurs in people with a structural heart disorder such as a heart attack, heart failure, or a cardiomyopathy. It is more common among older people. However, rarely, ventricular tachycardia develops in young people who do not have a structural heart disorder. Such young people may have a heart disorder called long QT syndrome, which can be inherited or caused by certain drugs. It can also be due to other rare inherited disorders such as the Brugada syndrome (a cardiac channelopathy).
People with ventricular tachycardia almost always have awareness of heart beats (palpitations). They may have weakness, light-headedness, and/or chest discomfort.
Sustained ventricular tachycardia can be dangerous because the ventricles cannot fill adequately or pump blood normally. Blood pressure tends to fall, and heart failure follows. Sustained ventricular tachycardia is also dangerous because it can worsen until it becomes ventricular fibrillation—a form of cardiac arrest. Sometimes ventricular tachycardia causes few symptoms, even at rates of up to 200 beats per minute, but it may still be extremely dangerous.
Electrocardiography (ECG) is used to diagnose ventricular tachycardia and to help determine whether treatment is required.
Ventricular tachycardia is treated when it causes symptoms or when episodes last more than 30 seconds even without causing symptoms.
People who have symptoms, particularly if blood pressure is too low, require immediate cardioversion (an electrical shock to convert the heart to normal rhythm).
People who have no symptoms but who have had ventricular tachycardia for more than 30 seconds should be treated either with cardioversion or intravenous drugs.
Cardioversion is painful, so sedation is required, but cardioversion is almost always effective and aside from the discomfort has few side effects.
Drugs are not uncomfortable but are not as effective as cardioversion in stopping the abnormal heart rhythm (arrhythmia) and are more likely to cause side effects. The most commonly used drugs are amiodarone, lidocaine, and procainamide (see table Some Drugs Used to Treat Arrhythmias).
The long-term goal is to prevent sudden death, rather than simply stopping the abnormal rhythm. In people with ventricular tachycardia who have an underlying heart disorder, particularly if their heart does not pump well, an implantable cardioverter-defibrillator (ICD, a small device that can detect an arrhythmia and deliver a shock to correct it) is often used. This procedure is similar to implantation of an artificial pacemaker.
Certain procedures may be used to destroy the small abnormal area in the ventricles, identified by ECG, that is usually responsible for sustained ventricular tachycardia. They include catheter ablation (delivery of energy of a specific frequency or cold through a catheter inserted in the heart) and open-heart surgery.
The following is an English-language resource that may be useful. Please note that THE MANUAL is not responsible for the content of this resource.
American Heart Association: Arrhythmia: Information to help people understand their risks of arrhythmias as well as information on diagnosis and treatment